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Clonal heterogeneity as a driver of disease variability in the evolution of myeloproliferative neoplasms.
OAI: oai:www.repository.cam.ac.uk:1810/245921
Abstract
Animals Cell Division Cell Transformation Neoplastic Clone Cells Cytokines Epigenesis Genetic evolution Molecular Gene Dosage Gene Knock-In Techniques Genetic Heterogeneity Hematopoietic Stem Cells Humans Janus Kinase 2 Leukemia Myeloid Models Genetic Multipotent Stem Cells Mutation Missense Myeloproliferative Disorders Neoplasm Proteins Neoplastic Stem Cells Phenotype Point Mutation RNA Splicing selection Genetic Signal Transduction Transcription Factors